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I have an application in which I need to implement functionality to print PDF file which is stored in folder in root directory. How can I achieve that ... Peroneal Muscular Atrophy [Charcot-Marie-Tooth Disease, or CMT Types 1 and 2; Hereditary Motor-Sensory Neuropathy, or HMSN Types I and II (Dyck); and Related Conditions (See Table 46-6)] The hallmarks of this class of polyneuropathies are genetic transmission, complete symmetry, slow progression, and loss of myelin out of proportion to axons These are the most common forms of inherited peripheral neuropathy and indeed among the most common of all inherited neurologic diseases The frequency of the disease cannot be stated with precision because of its clinical heterogeneity, but the usually quoted prevalence is 1 in 2500 of the population, the most frequent subtype occurring in 1 in 4000 More than 15 subtypes have been described but only a few occur with any regularity Because the disease was described in 1886 almost simultaneously by Tooth in England and by Charcot and Marie in France, all their names have been attached to it, even though similar cases had been recorded earlier by Eulenberg (1856), Friedreich (1873), Ormerod (1884), and Osler (1880) The secondary changes in the spinal cord and the occasional association of this disease with Friedreich ataxia led early observers to consider it as a hereditary myelopathy and not to class it with the neuropathies, but the evidence supporting the latter grouping is now unassailable The two important advances in our understanding of this disease since the original descriptions have been the separation of the main subtypes on the basis of their electrophysiologic (EMG) features, and the discovery of gene mutations which cause many of these diseases In most cases, these mutations can be related to alterations in components of peripheral nerve myelin or axons Genetic Transmission The pattern of inheritance of Charcot-Marie-Tooth disease is most often that of an autosomal dominant trait, with almost complete penetrance; less often it is autosomal recessive and rarely X-linked dominant or X-linked recessive (see Table 46-6) Some cases, probably a small number, arise as de novo mutations (Hoogendijk et al) The common types that are connected to chromosomes 1 or 17 cannot be easily distinguished from one another on clinical grounds but they have distinctive EMG features Seventy percent of CMT type 1 cases result from duplication of the gene for a peripheral myelin protein (PMP22) on chromosome 17 p11 (type CMT1A) Other studies of the PMP22 and PO (another myelin protein) gene expression in CMT1 and CMT3 cases (Dejerine-Sottas disease) have yielded discordant results because mutations on different loci, including one on chromosome 1, lead to different presentations, as noted in Table 46-6 The disease termed hereditary liability to pressure palsies also displays an aberration on chromosome 17, but in the form of a deletion rather than a duplication of the gene for PMP22 This disease is discussed further on, under Brachial Plexus Neuropathies Yet an additional X-linked variant is the result of a mutation of the gene for connexin-32, another component of myelin, and is seen reasonably often Undoubtedly, further studies of genes and gene products will continue to advance our understanding of the inherited neuropathies, but for the present, clinicians are still best served by the descriptive clinical classi cation outlined in Table 46-2 acknowledging, of course, the clinical heterogeneity of this class of disorders Clinical and Electrophysiologic Features Dyck and Lambert and Harding and Thomas have subdivided CMT disease into two. mvc open pdf in new tab How to create a PDF file in ASP.NET MVC - Syncfusion
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Embed an iframe inside a div pointing to Google Doc Viewer and specifying the PDF file you want to display . This is the code you should add:. I Charcot-Marie-Tooth (CMT) and related disorders 1 CMT1 (dominantly inherited myelinopathy) CMT1A* demyelinating CMT1B* CMT1C CMT1D, CMT4E CMT1F, CMT2E demyelinating Before leaving this discussion of MVRC, you should understand that Oracle has leveraged the internal processes and structures used to implement MVRC to add a set of flashback features to the database As the name implies, flashback is the ability to deliver a view of the database at an earlier point in time A number of flashback features are available in Oracle 11 g: Duplication of gene for peripheral myelin protein (PMP22) Myelin protein P0 Lipopolysaccharide-induced TNF Transcription factor EGR2 Neuro lament light subunit Rho guanine exchange factor Kinesin KIF1B motor protein Mitofusin (MFN2) GTP binding protein RAB7 10: Early are exia, distal weakness, sensory loss Early are exia, distal weakness, sensory loss May be severe and congenital demyelinating demyelinating demyelinating, axonal Congenital hypomyelination demyelinating 2 CMT2 (dominantly inherited axonopathy) CMT 2A* axonal CMT2A (alt) axonal CMT2B axonal CMT2C axonal CMT2D axonal CMT2F 1 axonal CMT2F 2 axonal 3 AR - CMT2 - (recessively inherited axonopathy) AR-CMT2A axonal 4 CMT3 (congenital myelinopathy) demyelinating 5 CMT4 (recessively inherited myelino-axonopathy) CMT4A demyelinating CMT4B CMT4B 2 CMT4C CMT4D CMT4F 6 CMT X (XD)* 7 Neuralgic amyotrophy (AD) 8 HNPP* demyelinating demyelinating demyelinating demyelinating demyelinating demyelinating axonal axonal devexpress asp.net mvc pdf viewer C# render pdf in browser using MVC - Tallcomponents
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